Hong Yang, M.D.
Research Assistant Professor
Department of Cardiovascular Biology ,
Department of Biomedical Sciences

(615) 327-5772
WBS Room 2007
1005 Dr. D.B. Todd Boulevard
Nashville, TN  37208

hyang@mmc.edu

Lab URL

CV or Bio (PDF)

Hubei Medical University, P. R. China. B.S. 1973 - 1976 Medicine
Hubei Medical University, P. R. China. M.D. 1983 - 1986 Cardiology/Physiology

Dr. Yang’s lab is investigating the mechanism of endothelial aging, especially the role of oxidative stress in age-related changes in endothelial nitric oxide metabolism, as well as the interaction of endothelial cells and leukocytes. The goal of research in her laboratory is to explore the proteins and pathways responsible for the age-related changes in endothelial functions, using proteomics techniques and molecular biology approaches.

Yang H, Deng C, Shen S, Zhang D, Yu Y.  Expression and significance of Bcl-2, Bax, Fas and caspase-3 in different phases of human hemangioma. J Huazhong Univ Sci Technolog Med Sci. 2006;26:402-4.

Yang H., L. J. Roberts, L.C. Zhou, M.J. Shi, B.R. Ballard, A. Richardson and Z.M. Guo. Retardation of atherosclerosis by overexpression of catalase or both Cu/Zn-superoxide and catalase in mice lacking apolipoprotein E. Cir. Res. 95:1075-1081, 2004.

Yang, H., M.J. Shi, J. Story, A. Richardson and Z.M. Guo. Food restriction attenuates age-related changes in mouse endothelial cell functions. J. Gerontol. Biol. Sci. 59:B316-B323, 2004.

Yang, H. M. Uddin, M.J. Shi, M. Polley-Mandal and Z.M. Guo. Elevation of oxidative stress in the aorta of genetically hypertensive mice. Mech. Aging Dev. 124:811—817, 2003.

Yang, H., M.J. Shi, H. VanRemmen, X.L. Chen, J. Vijg, A. Richardson and Z.M. Guo.  Overexpression of catalase in mice reduces the pressor response to vasoconstrictor agents.  A. J. Hypert. 16:1-5, 2003.2.